This panel provides drug-protein interaction and their ADRs along with references
Interacting Drugs |
Toxicity |
Mechanism |
Reference |
Dehydrated Alcohol | Impair Liver Regeneration | Ethanol treatment is associated with phosphatidylinositol-3 kinase with tyrosyl-phosphorylated IRS-1, and MAP kinase and phosphatidylinositol-3 kinase activities,suggesting that ethanol treatment substantially inhibits IRS-1 and MAP kinase signaling and growth-associated gene expression,which leads to chronic ethanol toxicity impairs liver regeneration, inhibits DNA synthesis, and mutes cellular responses to growth factor stimulation [ ADR Type 1 ] | Ethanol inhibition of insulin signaling in hepatocellular carcinoma cells
|
Dehydrated Alcohol | Inhibit Dna Synthesis | Ethanol treatment is associated with phosphatidylinositol-3 kinase with tyrosyl-phosphorylated IRS-1, and MAP kinase and phosphatidylinositol-3 kinase activities,suggesting that ethanol treatment substantially inhibits IRS-1 and MAP kinase signaling and growth-associated gene expression,which leads to chronic ethanol toxicity impairs liver regeneration, inhibits DNA synthesis, and mutes cellular responses to growth factor stimulation [ ADR Type 1 ] | Ethanol inhibition of insulin signaling in hepatocellular carcinoma cells
|
Dehydrated Alcohol | Mute Cellular Responses To Growth Factor Stimulation | Ethanol treatment is associated with phosphatidylinositol-3 kinase with tyrosyl-phosphorylated IRS-1, and MAP kinase and phosphatidylinositol-3 kinase activities,suggesting that ethanol treatment substantially inhibits IRS-1 and MAP kinase signaling and growth-associated gene expression,which leads to chronic ethanol toxicity impairs liver regeneration, inhibits DNA synthesis, and mutes cellular responses to growth factor stimulation [ ADR Type 1 ] | Ethanol inhibition of insulin signaling in hepatocellular carcinoma cells
|
This panel provides information on drug category